Amino Acids in Neurogenesis
Neuroscience is a fast-paced field. Like all medical disciplines to date, it is still primarily symptomatic oriented. Stimulants are being increasingly studied in Brain-signaling, rather than the initial precursors of all Signals, the Amino acids.
Amino acids are organic molecules virtually omnipresent in every living organism, the building blocks of life.
In the human body, they tend to bind in short chains named Peptides.
When Peptides contain at least 20 amino acids, they form Proteins.
Proteins are longer chains of amino acids also called Polypeptides.
A protein can have a structure of up to two Polypeptides.
In Mental Health, Proteins are outdated subjects of study.
Modafinil is the primary compound associated to protein synthesis to date, not yet understood after 30 years.
Coincidentally, this medication is somewhat ghosted, despite its effectiveness at cellular level.
Every biological process starts at cellular level.
Proteins play a crucial role in cellular development and Signal Processing, although pharmacology looks at the more remunerative symptomatic mechanisms of action.
Current stimulants target the manufacturing of dopamine and norepinephrine in ADHD and Narcolepsy.
They don’t target failing Brain Signaling, the root-cause of Executive Dysfunction.
Although the mechanism of action is ‘supposedly’ not understood 30 years later, Modafinil showed promising results with boosting Neural Transportation Pathways responsible for carrying signals from the Cerebellum to the Cerebrum by Proteinic synthesis.
For non-known reasons, research was archived and the compound is available as a non-specific source of evidence, therefore deemed unsafe and not promoted openly by professionals.
It is known that Tryptophan and Tyrosine are the precursors of Serotonin and Dopamine, respectively, the two main humoral neurotransmitters.
There is no medication targeting these proteins, other than over the counter’ supplements.
Research is bound towards Neurotransmission at Synaptic level only.
Signaling deficiency is not taken into consideration by pharmaceuticals.
What we supposedly don’t know is whether dysfunctional Neurotransmission is a Signal-disorder or a Re-uptake dysfunction.
Money will tell.
